Kanovsky, Mecheal
Articles
Michl, Josef;
Pincus, Matthew R.;
Friedman, Fred K.;
Kanovsky, Mecheal;
Botzolaki, Georgia;
Morin, Joseph;
Kovac, Cecilia;
Chung, Denise L.;
Chie, Lyndon, Peptides designed from molecular modeling studies of the ras-p21 protein induce phenotypic reversion of a pancreatic carcinoma cell line but have no effect on normal pancreatic acinar cell growth. Cancer Chemotherapy and Pharmacology 2003, 52(3), 202 - 208, 10.1007/s00280-003-0639-3 
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Michl, Josef;
Pincus, Matthew R.;
Brandt-Rauf, Paul W.;
Kanovsky, Mecheal;
Chung, Denise L.;
Chie, Lyndon;
Way, Denise;
Smith, Steven;
Sivendran, Sharmila, A protein kinase C inhibitor induces phenotypic reversion of ras-transformed pancreatic cancer cells and cooperatively blocks tumor cell proliferation with an anti-ras peptide. Cancer Chemotherapy and Pharmacology 2002, 49(6), 429 - 437, 10.1007/s00280-002-0432-8 .
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Michl, Josef;
Pincus, Matthew R.;
Friedman, Fred K.;
Brandt-Rauf, Paul W.;
Kanovsky, Mecheal;
Chie, Lyndon;
Smith, Steven;
Ranginwale, Masood;
Flom, Judy;
Chung, Denise;
Robinson, Richard C.;
Yamaizumi, Ziro, Differences in patterns of activation of MAP kinase induced by oncogenic ras-p21 and insulin in oocytes. Experimental Cell Research 2001, 269(1), 162 - 169, 10.1006/excr.2001.5311 .
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Michl, Josef;
Pincus, Matthew R.;
Friedman, Fred K.;
Fine, Robert L.;
Brandt-Rauf, Paul W.;
Rosal, Ramon;
Drew, Lisa;
Kanovsky, Mecheal;
Raffo, Anthony;
Do, Tamara;
Rubinstein, Pablo;
Visser, Jan;
Robinson, Richard, Peptides from the amino terminal mdm-2-binding domain of p53, designed from conformational analysis, are selectively cytotoxic to transformed cells. Proceedings of the National Academy of Sciences of the United States of America 2001, 98(22), 12438 - 12443, 10.1073/pnas.211280698 .
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